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Blockade of thromboxane A2 signaling attenuates ethanol-induced myocardial inflammatory response in mice.

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第一作者： Weilun,Ai

第一单位： Department of Internal Medicine, Division of Diabetes, Endocrinology, and Metabolism, University of Nebraska Medical Center, Omaha, Nebraska, USA.;VA Nebraska-Western Iowa Health Care System, Omaha, Nebraska, USA.

作者： Weilun,Ai ^[1] ; Carol A,Casey ^[2] ; Paras Kumar,Mishra ^[3] ; Yazen,Alnouti ^[4] ; Sohel,Daria ^[4] ; Viswanathan,Saraswathi ^[1]

作者单位： Department of Internal Medicine, Division of Diabetes, Endocrinology, and Metabolism, University of Nebraska Medical Center, Omaha, Nebraska, USA.;VA Nebraska-Western Iowa Health Care System, Omaha, Nebraska, USA. ^[1] VA Nebraska-Western Iowa Health Care System, Omaha, Nebraska, USA.;Department of Internal Medicine, Division of Gastroenterology and Hepatology, University of Nebraska Medical Center, Omaha, Nebraska, USA. ^[2] Department of Cellular and Integrative Physiology, University of Nebraska Medical Center, Omaha, Nebraska, USA. ^[3] Department of Pharmaceutical Sciences, College of Pharmacy, University of Nebraska Medical Center, Omaha, Nebraska, USA. ^[4]

关键词 alcohol alcohol‐associated cardiomyopathy inflammation myocardial remodeling thromboxane A2 receptor

DOI 10.1111/acer.15391

PMID 39030742

发布时间 2025-11-27

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Alcohol, clinical & experimental research

2024年48卷8期

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Blockade of thromboxane A2 signaling attenuates ethanol-induced myocardial inflammatory response in mice.

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Blockade of thromboxane A2 signaling attenuates ethanol-induced myocardial inflammatory response in mice.

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