SLC2A1 boosts the resistance of non-small cell lung cancer to taxanes by stimulating the formation of EPCAM<sup>+</sup> cancer stem-like cells via glycolysis.

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作者： Zhe,Yu ^[1] ; Jian,Sun ^[2] ; Kai,Fang ^[3] ; Jingwei,Xu ^[4] ; Jian,Yang ^[4] ; Dai,Chunlei ^[4] ; Yongsheng,Gong ^[4] ; Haitao,Ma ^[1]

作者单位： Department of Thoracic Surgery, The First Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China. ^[1] Center for Reproduction and Genetics, Suzhou Municipal Hospital &The Affiliated Suzhou Hospital of Nanjing Medical University, Suzhou, Jiangsu, China. ^[2] Department of Oncological Surgery, The Affiliated Hospital of Jiangnan University, Wuxi, Jiangsu province, China. ^[3] Department of Thoracic Surgery, Suzhou Municipal Hospital & The Affiliated Suzhou Hospital of Nanjing Medical University, Suzhou, Jiangsu, China. ^[4]

关键词 Chemo-resistance SLC2A1 Taxanes Tumor microenvironment

DOI 10.1016/j.tranon.2024.102082

PMID 39126936

发布时间 2024-09-09

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Translational oncology

2024年49卷

102082页

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SLC2A1 boosts the resistance of non-small cell lung cancer to taxanes by stimulating the formation of EPCAM<sup>+</sup> cancer stem-like cells via glycolysis.

Translational oncology

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Translational oncology

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SLC2A1 boosts the resistance of non-small cell lung cancer to taxanes by stimulating the formation of EPCAM<sup>+</sup> cancer stem-like cells via glycolysis.

Translational oncology

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Translational oncology

相关期刊

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