换一批STC2 knockdown inhibits cell proliferation and glycolysis in hepatocellular carcinoma through promoting autophagy by PI3K/Akt/mTOR pathway.
第一作者：
Ding,Li
第一单位：
Department of Interventional Radiology and Vascular Surgery, Hunan Provincial People's Hospital (the First Affiliated Hospital of Hunan Normal University), Changsha, 410005, Hunan, China.
作者：
医学主题词
癌, 肝细胞(Carcinoma, Hepatocellular);肝肿瘤(Liver Neoplasms);人类(Humans);细胞增殖(Cell Proliferation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);自噬(Autophagy);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);糖蛋白类(Glycoproteins);糖酵解(Glycolysis);信号传导(Signal Transduction);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);细胞系, 肿瘤(Cell Line, Tumor);动物(Animals);基因敲低技术(Gene Knockdown Techniques);小鼠(Mice);男(雄)性(Male);小鼠, 裸(Mice, Nude);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);小鼠, 近交BALB C(Mice, Inbred BALB C);细胞运动(Cell Movement)
DOI
10.1016/j.abb.2024.110149
PMID
39271096
发布时间
2026-05-16
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