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首页> Steroids> Neurotoxic mechanisms of dexamethasone in SH-SY5Y neuroblastoma cells: Insights into bioenergetics, oxidative stress, and apoptosis.

Neurotoxic mechanisms of dexamethasone in SH-SY5Y neuroblastoma cells: Insights into bioenergetics, oxidative stress, and apoptosis.

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作者： Ekramy M,Elmorsy ^[1] ; Ayat B,Al-Ghafari ^[2] ; Huda A,Al Doghaither ^[3] ; Manal S,Fawzy ^[4] ; Shaimaa A,Shehata ^[5]

作者单位： Pathology Department, Faculty of Medicine, Northern Border University, Arar 91431, Saudi Arabia; Center for Health Research, Northern Border University, Arar, Saudi Arabia. ^[1] Department of Biochemistry, Faculty of Science, King Abdulaziz University, Jeddah 21589, Saudi Arabia; Experimental Biochemistry Unit, King Fahd Medical Research Center, King Abdulaziz University, Jeddah 21589, Saudi Arabia. ^[2] Department of Biochemistry, Faculty of Science, King Abdulaziz University, Jeddah 21589, Saudi Arabia. ^[3] Department of Biochemistry, Faculty of Medicine, Northern Border University, Arar 91431, Saudi Arabia; Department of Medical Biochemistry and Molecular Biology, Faculty of Medicine, Suez Canal University, Ismailia 41522, Egypt. Electronic address: manal.darwish@nbu.edu.sa. ^[4] Department of Forensic Medicine and Clinical Toxicology, Faculty of Medicine, Suez Canal University, Ismailia 41522, Egypt. ^[5]

关键词 ATP Antimycin A (mitochondrial complex III inhibitor)Apoptosis Azide DEX Dexamethasone (DEX)Dichloroindophenol FCCP (Uncoupler)Glutathione (GSH)Hydrogen peroxide (H(2)O(2))Lactate Mitochondrial damage Neurotoxicity Oxidative stress Phosphate-buffered saline Reactive oxygen species (ROS)Rotenone (mitochondrial complex I inhibitor)

主题词人类(Humans);地塞米松(Dexamethasone);氧化性应激(Oxidative Stress);细胞凋亡(Apoptosis);神经母细胞瘤(Neuroblastoma);能量代谢(Energy Metabolism);细胞系, 肿瘤(Cell Line, Tumor);细胞存活(Cell Survival)

DOI 10.1016/j.steroids.2024.109514

PMID 39303897

发布时间 2024-11-21

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