MTAP protein status is highly concordant with CDKN2A fluorescent in situ hybridization and allows stratification of the luminal subtype in muscle-invasive bladder cancer.

导出 LinkOut

翻译打印收藏纠错

作者： Ekaterina,Olkhov-Mitsel ^[1] ; Alexander,Oberc ^[2] ; Kenneth J,Craddock ^[2] ; Christopher,Sherman ^[2] ; Elzbieta,Slodkowska ^[2] ; Michelle R,Downes ^[2]

作者单位： Division of Anatomic Pathology, Department of Laboratory Medicine and Molecular Diagnostics, Precision Diagnostics and Therapeutics Program, Sunnybrook Health Sciences Centre, Toronto, ON, Canada. ^[1] Division of Anatomic Pathology, Department of Laboratory Medicine and Molecular Diagnostics, Precision Diagnostics and Therapeutics Program, Sunnybrook Health Sciences Centre, Toronto, ON, Canada.;Laboratory Medicine and Pathobiology, University of Toronto, Toronto, ON, Canada. ^[2]

关键词 9p21 loss CDKN2A deletions MTAP loss metastatic urothelial carcinoma molecular subtyping muscle‐invasive bladder cancer p16 prognostic biomarkers therapeutic targets

DOI 10.1111/his.15324

PMID 39327852

发布时间 2024-09-27

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Histopathology

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

MTAP protein status is highly concordant with CDKN2A fluorescent in situ hybridization and allows stratification of the luminal subtype in muscle-invasive bladder cancer.

Histopathology

相似文献

Histopathology

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

MTAP protein status is highly concordant with CDKN2A fluorescent in situ hybridization and allows stratification of the luminal subtype in muscle-invasive bladder cancer.

Histopathology

相似文献

Histopathology

相关期刊

检索历史清除