首页> The FEBS journal> Hemin-induced platelet activation is regulated by the ACKR3 chemokine surface receptor and has implications for passivation of vulnerable atherosclerotic plaques.

Hemin-induced platelet activation is regulated by the ACKR3 chemokine surface receptor and has implications for passivation of vulnerable atherosclerotic plaques.

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第一作者： Zoi,Laspa

第一单位： Department of Cardiology and Angiology, University Hospital Tübingen, University Tübingen, Germany.

作者： Zoi,Laspa ^[1] ; Valerie,Dicenta-Baunach ^[1] ; David,Schaale ^[1] ; Manuel,Sigle ^[1] ; Ravi,Hochuli ^[1] ; Tatsiana,Castor ^[1] ; Alp,Bayrak ^[2] ; Tobias,Harm ^[1] ; Karin Anne Lydia,Müller ^[1] ; Thanigaimalai,Pillaiyar ^[3] ; Stefan,Laufer ^[4] ; Anne-Katrin,Rohlfing ^[1] ; Meinrad Paul,Gawaz ^[1]

作者单位： Department of Cardiology and Angiology, University Hospital Tübingen, University Tübingen, Germany. ^[1] Department of Pharmaceutical and Medicinal Chemistry, Institute of Pharmaceutical Sciences, University Tübingen, Germany. ^[2] Department of Pharmaceutical and Medicinal Chemistry, Institute of Pharmaceutical Sciences, University Tübingen, Germany.;Tübingen Center for Academic Drug Discovery & Development (TüCAD2), Germany. ^[3] Department of Pharmaceutical and Medicinal Chemistry, Institute of Pharmaceutical Sciences, University Tübingen, Germany.;Tübingen Center for Academic Drug Discovery & Development (TüCAD2), Germany.;iFIT Cluster of Excellence EXC 2180 'Image-Guided and Functionally Instructed Tumor Therapies', University of Tübingen, Germany. ^[4]

关键词 ACKR3 hemin intraplaque hemorrhage (IPH)multi‐color flow cytometry platelets