Controlled infection with cryopreserved human hookworm induces CTLA-4 expression on Tregs and upregulates tryptophan metabolism.

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作者： Francesco,Vacca ^[1] ; Thomas C,Mules ^[2] ; Mali,Camberis ^[1] ; Brittany,Lavender ^[1] ; Sophia-Louise,Noble ^[3] ; Alissa,Cait ^[1] ; Kate,Maclean ^[1] ; John,Mamum ^[1] ; Bibek,Yumnam ^[1] ; Tama,Te Kawa ^[1] ; Laura,Ferrer-Font ^[4] ; Jeffry S,Tang ^[1] ; Olivier,Gasser ^[1] ; Graham,Le Gros ^[1] ; Stephen,Inns ^[5]

作者单位： Le Gros Laboratory, Malaghan Institute of Medical Research, Wellington, New Zealand. ^[1] Le Gros Laboratory, Malaghan Institute of Medical Research, Wellington, New Zealand.;Department of Medicine, University of Otago, Wellington, New Zealand.;Gastroenterology, Te Whatu Ora, Capital Coast and Hutt Valley, Wellington, New Zealand. ^[2] Le Gros Laboratory, Malaghan Institute of Medical Research, Wellington, New Zealand.;Department of Medicine, University of Otago, Wellington, New Zealand. ^[3] Hugh Green Technology Centre, Malaghan Institute of Medical Research, Wellington, New Zealand. ^[4] Department of Medicine, University of Otago, Wellington, New Zealand.;Gastroenterology, Te Whatu Ora, Capital Coast and Hutt Valley, Wellington, New Zealand. ^[5]

关键词 Hookworm clinical trial controlled infection cryopreservation eosinophilia helminths immune regulation microbiome

主题词人类(Humans);T淋巴细胞, 调节性(T-Lymphocytes, Regulatory);色氨酸(Tryptophan);动物(Animals);低温保存(Cryopreservation);钩虫感染(Hookworm Infections);幼虫(Larva);增量调节(Up-Regulation);成年人(Adult);女(雌)性(Female);粪便(Feces);钩口总科(Ancylostomatoidea);男(雄)性(Male)

DOI 10.1080/19490976.2024.2416517

PMID 39411786

发布时间 2024-10-19

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Controlled infection with cryopreserved human hookworm induces CTLA-4 expression on Tregs and upregulates tryptophan metabolism.

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Controlled infection with cryopreserved human hookworm induces CTLA-4 expression on Tregs and upregulates tryptophan metabolism.

Gut microbes

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Gut microbes

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