首页> Gastroenterology report> Cooperation of Wnt/β-catenin and Dll1-mediated Notch pathway in Lgr5-positive intestinal stem cells regulates the mucosal injury and repair in DSS-induced colitis mice model.

Cooperation of Wnt/β-catenin and Dll1-mediated Notch pathway in Lgr5-positive intestinal stem cells regulates the mucosal injury and repair in DSS-induced colitis mice model.

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第一作者： Weijun,Ou

第一单位： Department of Colorectal Surgery, Xinhua Hospital, Shanghai Jiaotong University, School of Medicine, Shanghai, P. R. China.;Shanghai Colorectal Cancer Research Center, Shanghai, P. R. China.

作者： Weijun,Ou ^[1] ; Weimin,Xu ^[1] ; Yaosheng,Wang ^[1] ; Zhebin,Hua ^[1] ; Wenjun,Ding ^[1] ; Long,Cui ^[1] ; Peng,Du ^[1]

作者单位： Department of Colorectal Surgery, Xinhua Hospital, Shanghai Jiaotong University, School of Medicine, Shanghai, P. R. China.;Shanghai Colorectal Cancer Research Center, Shanghai, P. R. China. ^[1]

关键词 Lgr5 Notch intestinal stem cell ulcerative colitis β-catenin

DOI 10.1093/gastro/goae090

PMID 39444950

发布时间 2024-10-25

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Cooperation of Wnt/β-catenin and Dll1-mediated Notch pathway in Lgr5-positive intestinal stem cells regulates the mucosal injury and repair in DSS-induced colitis mice model.

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Cooperation of Wnt/β-catenin and Dll1-mediated Notch pathway in Lgr5-positive intestinal stem cells regulates the mucosal injury and repair in DSS-induced colitis mice model.

Gastroenterology report

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Gastroenterology report

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