Epoxytiglianes induce keratinocyte wound healing responses via classical protein kinase C activation to promote skin re-epithelialization.

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第一作者： Rachael L,Moses

第一单位： Disease Mechanisms Group, School of Dentistry, College of Biomedical and Life Sciences, Cardiff University, UK; Melbourne Dental School, Faculty of Medicine, Dentistry and Health Sciences, University of Melbourne, Victoria, Australia.

作者： Rachael L,Moses ^[1] ; Emma L,Woods ^[2] ; Jordanna,Dally ^[2] ; Jenny P,Johns ^[3] ; Vera,Knäuper ^[2] ; Glen M,Boyle ^[3] ; Victoria,Gordon ^[4] ; Paul,Reddell ^[4] ; Robert,Steadman ^[5] ; Ryan,Moseley ^[6]

作者单位： Disease Mechanisms Group, School of Dentistry, College of Biomedical and Life Sciences, Cardiff University, UK; Melbourne Dental School, Faculty of Medicine, Dentistry and Health Sciences, University of Melbourne, Victoria, Australia. ^[1] Disease Mechanisms Group, School of Dentistry, College of Biomedical and Life Sciences, Cardiff University, UK. ^[2] Cancer Drug Mechanisms Group, QIMR Berghofer Medical Research Institute, Brisbane, Queensland, Australia. ^[3] QBiotics Group, Yungaburra, Queensland, Australia. ^[4] Wales Kidney Research Unit, Division of Infection and Immunity, School of Medicine, College of Biomedical and Life Sciences, Cardiff University, UK. ^[5] Disease Mechanisms Group, School of Dentistry, College of Biomedical and Life Sciences, Cardiff University, UK. Electronic address: MoseleyR@cardiff.ac.uk. ^[6]

关键词 Epoxytigliane Keratinocyte Migration Proliferation Protein kinase C Re-epithelialization