The Role of Microhomology-Mediated End Joining (MMEJ) at Dysfunctional Telomeres.

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第一作者： David,Billing

第一单位： Department of Radiation Oncology, Memorial Sloan Kettering Cancer Center, New York, New York 10065, USA.;Molecular Biology Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York 10065, USA.

作者： David,Billing ^[1] ; Agnel,Sfeir ^[2]

作者单位： Department of Radiation Oncology, Memorial Sloan Kettering Cancer Center, New York, New York 10065, USA.;Molecular Biology Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York 10065, USA. ^[1] Molecular Biology Program, Sloan Kettering Institute, Memorial Sloan Kettering Cancer Center, New York, New York 10065, USA sfeira@mskcc.org. ^[2]

主题词端粒(Telomere);人类(Humans);DNA断裂, 双链(DNA Breaks, Double-Stranded);动物(Animals)

DOI 10.1101/cshperspect.a041687

PMID 39500624

发布时间 2025-02-03

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准确一般字义偏差语意偏离译文作用小提交