Lactate activates ER stress to promote alveolar epithelial cells apoptosis in pulmonary fibrosis.

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作者： Zhiheng,Sun ^[1] ; Wanyu,He ^[2] ; Huiwen,Meng ^[2] ; Zhihua,Ji ^[2] ; Junxing,Qu ^[3] ; Guoying,Yu ^[4]

作者单位： College of Life Science, Institute of Biomedical Science, Henan Normal University, Xinxiang, Henan, China. sunzhiheng@htu.edu.cn.;State Key Laboratory of Cell Differentiation and Regulation, Xinxiang, Henan, China. sunzhiheng@htu.edu.cn. ^[1] College of Life Science, Institute of Biomedical Science, Henan Normal University, Xinxiang, Henan, China.;State Key Laboratory of Cell Differentiation and Regulation, Xinxiang, Henan, China. ^[2] Institutes of Health Central Plains, Xinxiang Medical University, Xinxiang, Henan, China. qujx@xxmu.edu.cn.;Xinxiang Key Laboratory for Tumor Drug Screening and Targeted Therapy, Xinxiang, Henan, China. qujx@xxmu.edu.cn. ^[3] College of Life Science, Institute of Biomedical Science, Henan Normal University, Xinxiang, Henan, China. guoyuying@htu.edu.cn.;State Key Laboratory of Cell Differentiation and Regulation, Xinxiang, Henan, China. guoyuying@htu.edu.cn. ^[4]

关键词 Alveolar epithelial cells Apoptosis Caspase-12 Endoplasmic reticulum stress Lactate Pulmonary fibrosis