Augmenting antitumor efficacy of Th17-derived Th1 cells through IFN-γ-induced type I interferon response network via IRF7.

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作者： Xiaoyi,Lei ^[1] ; Ruipei,Xiao ^[1] ; Zhe,Chen ^[1] ; Jie,Ren ^[1] ; Wenli,Zhao ^[1] ; Wenting,Tang ^[2] ; Kang,Wen ^[3] ; Yihan,Zhu ^[1] ; Xinru,Li ^[1] ; Suidong,Ouyang ^[4] ; Abai,Xu ^[3] ; Yu,Hu ^[2] ; Enguang,Bi ^[5]

作者单位： Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Guangdong Provincial Key Laboratory of Single Cell Technology and Application, Southern Medical University, Guangzhou, Guangdong 510515, China. ^[1] Division of Nephrology, State Key Laboratory of Organ Failure Research, National Clinical Research Center of Kidney Disease, Guangdong Provincial Institute of Nephrology, Guangdong Provincial Key Laboratory of Renal Failure Research, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China. ^[2] Department of Urology, Zhujiang Hospital, Southern Medical University, Guangzhou 510280, China. ^[3] Guangdong Provincial Key Laboratory of Medical Molecular Diagnostics, Guangdong Medical University, Dongguan 523808, China. ^[4] Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences, Guangdong Provincial Key Laboratory of Single Cell Technology and Application, Southern Medical University, Guangzhou, Guangdong 510515, China.;Department of Urology, Zhujiang Hospital, Southern Medical University, Guangzhou 510280, China. ^[5]

关键词 T helper cells adoptive cell transfer exhaustion tumor immunotherapy type I interferon pathway