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Scavenger Receptor CD36 in Tumor-Associated Macrophages Promotes Cancer Progression by Dampening Type-I IFN Signaling.

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第一作者： Ziyan,Xu

第一单位： NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, California.;School of Biological Sciences, University of California San Diego, La Jolla, California.

作者： Ziyan,Xu ^[1] ; Alexandra,Kuhlmann-Hogan ^[2] ; Shihao,Xu ^[2] ; Hubert,Tseng ^[2] ; Dan,Chen ^[2] ; Shirong,Tan ^[2] ; Ming,Sun ^[2] ; Victoria,Tripple ^[2] ; Marcus,Bosenberg ^[3] ; Kathryn,Miller-Jensen ^[4] ; Susan M,Kaech ^[2]

作者单位： NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, California.;School of Biological Sciences, University of California San Diego, La Jolla, California. ^[1] NOMIS Center for Immunobiology and Microbial Pathogenesis, Salk Institute for Biological Studies, La Jolla, California. ^[2] Department of Pathology, Yale School of Medicine, New Haven, Connecticut.;Department of Dermatology, Yale School of Medicine, New Haven, Connecticut.;Yale Center for Precision Cancer Modeling, Yale School of Medicine, New Haven, Connecticut.;Department of Immunobiology, Yale School of Medicine, New Haven, Connecticut. ^[3] Department of Biomedical Engineering, Yale University, New Haven, Connecticut.;Department of Molecular, Cellular and Developmental Biology, Yale University, New Haven, Connecticut. ^[4]