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Cyclin E1/CDK2 activation defines a key vulnerability to WEE1 kinase inhibition in gynecological cancers.

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第一作者: Daehwan,Kim
第一单位: Zentalis Pharmaceuticals, Inc., San Diego, CA, USA.
作者单位: Zentalis Pharmaceuticals, Inc., San Diego, CA, USA. [1] Division of Oncological Sciences, Knight Cancer Institute, Oregon Health and Science University, Portland, OR, USA. [2] Division of Oncological Sciences, Knight Cancer Institute, Oregon Health and Science University, Portland, OR, USA.;Cancer Early Detection Advanced Research Center (CEDAR), Knight Cancer Institute, Oregon Health and Science University, Portland, OR, USA. [3] Program in Women's Oncology, Women & Infants Hospital, Legorreta Cancer Center of Alpert Medical School at Brown University, Providence, RI, USA. [4] Early Phase Trials Unit, Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. [5] The University of Chicago Medicine & Biological Sciences, Chicago, IL, USA. [6] Ovarian Cancer Research Center, Division of Gynecologic Oncology, Department of Obstetrics & Gynecology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA. [7] Department of Investigational Cancer Therapeutics, Division of Cancer Medicine, University of Texas MD Anderson Cancer Center, Houston, TX, USA. [8] Zentalis Pharmaceuticals, Inc., San Diego, CA, USA. jma@zentalis.com. [9] Zentalis Pharmaceuticals, Inc., San Diego, CA, USA. mlackner@zentalis.com. [10]
DOI 10.1038/s41698-024-00787-4
PMID 39755818
发布时间 2025-01-07
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NPJ precision oncology

NPJ precision oncology

2025年9卷1期

3页

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