Loss of PKM2 dysregulates inflammatory signaling in the infarcted murine heart.

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作者： Katie C Y,Lee ^[1] ; Allison L,Williams ^[2] ; Akitoshi,Hara ^[2] ; Vedbar S,Khadka ^[3] ; Jeffrey,Hayashi ^[4] ; Ralph V,Shohet ^[2]

作者单位： Department of Medicine, John A. Burns School of Medicine, University of Hawaii Mānoa, Honolulu, Hawaii, USA.;Department of Cell and Molecular Biology, John A. Burns School of Medicine, University of Hawaii Mānoa, Honolulu, Hawaii, USA. ^[1] Department of Medicine, John A. Burns School of Medicine, University of Hawaii Mānoa, Honolulu, Hawaii, USA. ^[2] Bioinformatics Core, Department of Quantitative Health Sciences, John A. Burns School of Medicine, University of Hawaii Mānoa, Honolulu, Hawaii, USA. ^[3] John A. Burns School of Medicine, University of Hawaii Mānoa, Honolulu, Hawaii, USA. ^[4]

关键词 fibrosis inflammation myocardial infarction oxidative stress

主题词动物(Animals);心肌梗死(Myocardial Infarction);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);男(雄)性(Male);丙酮酸激酶(Pyruvate Kinase);信号传导(Signal Transduction);氧化性应激(Oxidative Stress);小鼠, 近交C57BL(Mice, Inbred C57BL);炎症(Inflammation);心肌(Myocardium);巨噬细胞(Macrophages);甲状腺激素类(Thyroid Hormones);白细胞介素6(Interleukin-6)

DOI 10.14814/phy2.70193

PMID 39761962

发布时间 2025-01-06

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Physiological reports

2025年13卷1期

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Loss of PKM2 dysregulates inflammatory signaling in the infarcted murine heart.

Physiological reports

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Loss of PKM2 dysregulates inflammatory signaling in the infarcted murine heart.

Physiological reports

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Physiological reports

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