Regulation of fibronectin and collagens type I, III and VI by TNF-α, TGF-β, IL-13, and tofacitinib.

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作者： Frederik S,Gillesberg ^[1] ; Martin,Pehrsson ^[2] ; Anne-Christine,Bay-Jensen ^[2] ; Peder,Frederiksen ^[2] ; Morten,Karsdal ^[2] ; Bent W,Deleuran ^[3] ; Tue W,Kragstrup ^[4] ; Satoshi,Kubo ^[5] ; Yoshiya,Tanaka ^[5] ; Joachim H,Mortensen ^[2]

作者单位： Nordic Bioscience, Immunoscience, Herlev Hovedgade 205-207, Herlev, 2730, Denmark. frg@nordicbio.com.;Department of Biomedicine, Aarhus University, Høegh-Guldbergs Gade 10, Aarhus C, 8000, Denmark. frg@nordicbio.com. ^[1] Nordic Bioscience, Immunoscience, Herlev Hovedgade 205-207, Herlev, 2730, Denmark. ^[2] Department of Biomedicine, Aarhus University, Høegh-Guldbergs Gade 10, Aarhus C, 8000, Denmark.;Department of Rheumatology, Århus University Hospital, Palle Juul-Jensens Boulevard 99, Aarhus N, 8200, Denmark. ^[3] Department of Biomedicine, Aarhus University, Høegh-Guldbergs Gade 10, Aarhus C, 8000, Denmark.;Department of Rheumatology, Århus University Hospital, Palle Juul-Jensens Boulevard 99, Aarhus N, 8200, Denmark.;Diagnostic Center, Regional Hospital Silkeborg, Falkevej 1, Silkeborg, 8600, Denmark. ^[4] The First Department of Internal Medicine, School of Medicine, University of Occupational & Environmental Health, Japan, 1-1 Iseigaoka, Yahata-nishi, 807-8555, Kitakyushu, Japan. ^[5]

关键词 Collagen Fibro-inflammation Fibrosis JAK inhibitor Tofacitinib Wound healing