Interleukin-6 is significantly increased in severe pneumonia after allo-HSCT and might induce lung injury via IL-6/sIL-6R/JAK1/STAT3 pathway.

导出 LinkOut

翻译打印收藏纠错

第一作者： Jing-Rui,Zhou

第一单位： Peking University People's Hospital, Peking University Institute of Hematology, National Clinical Research Center for Treatment of Hematological Disease, Beijing Key Laboratory of Hematopoietic Stem Cell Transplantation for the Treatment of Hematological Diseases, Beijing, China.

作者： Jing-Rui,Zhou ^[1] ; Yi,Liao ^[1] ; Le-Qing,Cao ^[1] ; Rui,Ma ^[1] ; Yun,He ^[1] ; Na,Li ^[1] ; Dan-Ping,Zhu ^[1] ; Xiao-Su,Zhao ^[1] ; Xiao-Jun,Huang ^[2] ; Yu-Qian,Sun ^[1]

作者单位： Peking University People's Hospital, Peking University Institute of Hematology, National Clinical Research Center for Treatment of Hematological Disease, Beijing Key Laboratory of Hematopoietic Stem Cell Transplantation for the Treatment of Hematological Diseases, Beijing, China. ^[1] Peking University People's Hospital, Peking University Institute of Hematology, National Clinical Research Center for Treatment of Hematological Disease, Beijing Key Laboratory of Hematopoietic Stem Cell Transplantation for the Treatment of Hematological Diseases, Beijing, China.;Peking-Tsinghua Center for Life Sciences, Academy for Advanced Interdisciplinary Studies, Peking University, Beijing, China. ^[2]

关键词 Interleukin 6 acute lung injury allo-HSCT endothelial cell dysfunction inflammation severe pneumonia stem cell transplantation

DOI 10.1093/infdis/jiaf041

PMID 39828908

发布时间 2025-01-20

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

The Journal of infectious diseases

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Interleukin-6 is significantly increased in severe pneumonia after allo-HSCT and might induce lung injury via IL-6/sIL-6R/JAK1/STAT3 pathway.

The Journal of infectious diseases

相似文献

The Journal of infectious diseases

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Interleukin-6 is significantly increased in severe pneumonia after allo-HSCT and might induce lung injury via IL-6/sIL-6R/JAK1/STAT3 pathway.

The Journal of infectious diseases

相似文献

The Journal of infectious diseases

相关期刊

检索历史清除