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WWC proteins-mediated compensatory mechanism restricts schwannomatosis driven by <i>NF2</i> loss of function.

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第一作者： Xueying,Wang

第一单位： Institute of Pediatrics, Children's Hospital of Fudan University, and Shanghai Key Laboratory of Medical Epigenetics, International Co-laboratory of Medical Epigenetics and Metabolism, State Key Laboratory of Genetic Engineering, Institutes of Biomedical Sciences, Shanghai Medical College, Fudan University, Shanghai, China.

作者： Xueying,Wang ^[1] ; Rui,Zhu ^[1] ; Pengcheng,Yu ^[1] ; Sixian,Qi ^[1] ; Zhenxing,Zhong ^[1] ; Ruxin,Jin ^[1] ; Yu,Wang ^[1] ; Yuan,Gu ^[1] ; Dan,Ye ^[2] ; Kang,Chen ^[3] ; Yilai,Shu ^[4] ; Yi,Wang ^[5] ; Fa-Xing,Yu ^[1]

作者单位： Institute of Pediatrics, Children's Hospital of Fudan University, and Shanghai Key Laboratory of Medical Epigenetics, International Co-laboratory of Medical Epigenetics and Metabolism, State Key Laboratory of Genetic Engineering, Institutes of Biomedical Sciences, Shanghai Medical College, Fudan University, Shanghai, China. ^[1] Huashan Hospital and Institutes of Biomedical Sciences, Shanghai Medical College, Fudan University, Shanghai, China. ^[2] Department of Obstetrics and Gynecology and Barbara Ann Karmanos Cancer Institute, Wayne State University, Detroit, MI, USA. ^[3] ENT Institute and Otorhinolaryngology Department of Affiliated Eye and ENT Hospital, State Key Laboratory of Medical Neurobiology, Fudan University, Shanghai, China. ^[4] Department of Neurology, Children's Hospital of Fudan University, National Children's Medical Center, Fudan University, Shanghai, China. ^[5]

医学主题词动物(Animals);神经鞘瘤(Neurilemmoma);小鼠(Mice);神经纤维素2(Neurofibromin 2);神经纤维瘤病(Neurofibromatoses);泛素蛋白连接酶类(Ubiquitin-Protein Ligases);皮肤肿瘤(Skin Neoplasms);人类(Humans);疾病模型, 动物(Disease Models, Animal);许旺细胞(Schwann Cells);神经纤维瘤病2型(Neurofibromatosis 2);泛素化(Ubiquitination);突变(Mutation)

DOI 10.1126/sciadv.adp4765

PMID 39841844

发布时间 2025-05-02

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