Chlamydia muridarum Causes Persistent Subclinical Infection and Elicits Innate and Adaptive Immune Responses in C57BL/6J, BALB/cJ, and J:ARC(S) Mice Following Exposure to Shedding Mice.

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第一作者： Noah,Mishkin

第一单位： 1Tri-Institutional Training Program in Laboratory Animal Medicine and Science, Memorial Sloan Kettering Cancer Center, Weill Cornell Medicine, and The Rockefeller University, New York, New York.

作者： Noah,Mishkin ^[1] ; Sebastian E,Carrasco ^[2] ; Michael,Palillo ^[1] ; Panagiota,Momtsios ^[3] ; Cheryl,Woods ^[3] ; Kenneth S,Henderson ^[3] ; Ana Leda F,Longhini ^[4] ; Chelsea,Otis ^[4] ; Rui,Gardner ^[4] ; Ann M,Joseph ^[5] ; Gregory F,Sonnenberg ^[5] ; Jack,Palillo ^[6] ; Rodolfo J Ricart,Arbona ^[2] ; Neil S,Lipman ^[2]

作者单位： 1Tri-Institutional Training Program in Laboratory Animal Medicine and Science, Memorial Sloan Kettering Cancer Center, Weill Cornell Medicine, and The Rockefeller University, New York, New York. ^[1] 1Tri-Institutional Training Program in Laboratory Animal Medicine and Science, Memorial Sloan Kettering Cancer Center, Weill Cornell Medicine, and The Rockefeller University, New York, New York.;2Center of Comparative Medicine and Pathology, Memorial Sloan Kettering Cancer Center and Weill Cornell Medicine, New York, New York. ^[2] 3Research Animal Diagnostic Services, Charles River Laboratories, Wilmington, Massachusetts. ^[3] 4Flow Cytometry Core Facility, Memorial Sloan Kettering Cancer Center, New York, New York. ^[4] 5Joan and Sanford I. Weill Department of Medicine, Division of Gastroenterology and Hepatology, Weill Cornell Medicine, Cornell University, New York, New York; and. ^[5] 6Neurological Clinical Research Institute, Massachusetts General Hospital, Boston, Massachusetts. ^[6]