Super-Enhancer Protects Cells From Toxicity of C9orf72 Poly(proline-arginine) by Inducing the Expression of KPNA2/KPNB1.

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作者： Miaomiao,Chen ^[1] ; Henglu,Cui ^[2] ; Xiaoyu,Zhang ^[3] ; Shuyan,Ma ^[2] ; Jinjing,Guo ^[2] ; Zhaoxiu,Liu ^[3] ; Donghua,Gu ^[4] ; Yihui,Fan ^[2]

作者单位： Laboratory of Medical Science, School of Medicine, Nantong University, Nantong, China. ^[1] Laboratory of Medical Science, School of Medicine, Nantong University, Nantong, China.;Department of Pathogenic Biology, School of Medicine, Nantong University, Nantong, China. ^[2] Department of Gastroenterology, Medical School of Nantong University, Affiliated Hospital of Nantong University, Nantong, China. ^[3] The Department of Urology, The Second Affiliated Hospital of Nantong University, Nantong University, Nantong, Jiangsu, China. ^[4]

关键词 C9orf72 KPNA2 KPNB1 amyotrophic lateral sclerosis (ALS)dipeptide repeat (DPR)frontotemporal dementia (FTD)super‐enhancer

主题词人类(Humans);β核胞浆转运蛋白类(beta Karyopherins);α核胞浆转运蛋白类(alpha Karyopherins);肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis)

DOI 10.1002/cbf.70053

PMID 39891383

发布时间 2025-02-01

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