Loss of PRICKLE1 leads to abnormal endometrial epithelial architecture, decreased embryo implantation, and reduced fertility in mice.

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作者： Emily R,Roberts ^[1] ; Aishwarya V,Bhurke ^[2] ; Sornakala,Ganeshkumar ^[1] ; Sumedha,Gunewardena ^[3] ; Ripla,Arora ^[2] ; Vargheese M,Chennathukuzhi ^[1]

作者单位： Department of Cell Biology and Physiology, University of Kansas Medical Center, Kansas City, KS 66160, USA. ^[1] Department of Obstetrics, Gynecology, and Reproductive Biology, Michigan State University, Grand Rapids, MI 49503, USA.;Institute for Quantitative Health Science and Engineering, Michigan State University, East Lansing, MI 48824, USA. ^[2] Department of Cell Biology and Physiology, University of Kansas Medical Center, Kansas City, KS 66160, USA.;Department of Biostatistics, University of Kansas Medical Center, Kansas City, KS 66160, USA. ^[3]

关键词 EMT Prickle1 Wnt/PCP epithelial polarity implantation

DOI 10.1093/pnasnexus/pgaf024

PMID 39917256

发布时间 2025-02-11

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Loss of PRICKLE1 leads to abnormal endometrial epithelial architecture, decreased embryo implantation, and reduced fertility in mice.

PNAS nexus

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PNAS nexus

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Loss of PRICKLE1 leads to abnormal endometrial epithelial architecture, decreased embryo implantation, and reduced fertility in mice.

PNAS nexus

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PNAS nexus

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