Zinc regulates microglial polarization and inflammation through IKBα after spinal cord injury and promotes neuronal repair and motor function recovery in mice.

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作者： Daoyong,Li ^[1] ; Mingyu,Bai ^[1] ; Zhanpeng,Guo ^[2] ; Yang,Cui ^[1] ; Xifan,Mei ^[1] ; He,Tian ^[3] ; Zhaoliang,Shen ^[4]

作者单位： The Third Affiliated Hospital of Jinzhou Medical University, Jinzhou City, Liaoning, China.;Key Laboratory of Liaoning Medical Organization Engineering, Jinzhou, Liaoning, China. ^[1] Key Laboratory of Liaoning Medical Organization Engineering, Jinzhou, Liaoning, China. ^[2] Key Laboratory of Liaoning Medical Organization Engineering, Jinzhou, Liaoning, China.;School of Basic Medicine, Jinzhou Medical University, Jinzhou, Liaoning, China. ^[3] The Third Affiliated Hospital of Jinzhou Medical University, Jinzhou City, Liaoning, China. ^[4]

关键词 functional recovery inflammatory response microglial polarization spinal cord injury zinc

DOI 10.3389/fphar.2025.1510372

PMID 39944615

发布时间 2025-02-13

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Frontiers in pharmacology

2025年16卷

1510372页

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Zinc regulates microglial polarization and inflammation through IKBα after spinal cord injury and promotes neuronal repair and motor function recovery in mice.

Frontiers in pharmacology

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Frontiers in pharmacology

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Zinc regulates microglial polarization and inflammation through IKBα after spinal cord injury and promotes neuronal repair and motor function recovery in mice.

Frontiers in pharmacology

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Frontiers in pharmacology

相关期刊

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