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R406 and its structural analogs reduce SNCA/α-synuclein levels via autophagic degradation.

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第一作者： Chao,Zhong

第一单位： The State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, Huashan Hospital, School of Life Sciences, The Institutes of Brain Science, Fudan University, Shanghai, China.

作者： Chao,Zhong ^[1] ; Xiaoge,Gao ^[1] ; Qi,Chen ^[2] ; Bowen,Guan ^[1] ; Wanli,Wu ^[1] ; Zhiqiang,Ma ^[1] ; Mengdan,Tao ^[2] ; Xihuan,Liu ^[1] ; Yu,Ding ^[1] ; Yiyan,Fei ^[3] ; Yan,Liu ^[2] ; Boxun,Lu ^[1] ; Zhaoyang,Li ^[1]

作者单位： The State Key Laboratory of Medical Neurobiology and MOE Frontiers Center for Brain Science, Huashan Hospital, School of Life Sciences, The Institutes of Brain Science, Fudan University, Shanghai, China. ^[1] Institute for Stem Cell and Neural Regeneration, School of Pharmacy, State Key Laboratory of Reproductive Medicine, Key Laboratory of Targeted Intervention of Cardiovascular Disease, Collaborative Innovation Center for Cardiovascular Disease Translational Medicine, Nanjing Medical University, Nanjing, China. ^[2] Department of Optical Science and Engineering, Shanghai Engineering Research Center of Ultra-Precision Optical Manufacturing, Key Laboratory of Micro and Nano Photonic Structures (Ministry of Education), Fudan University, Shanghai, China. ^[3]

关键词 Autophagic degradation SNCA/synuclein alpha midbrain organoid parkinson disease small molecule compounds

DOI 10.1080/15548627.2025.2483886

PMID 40143425

发布时间 2025-04-04

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