Cooperation of a polymerizing SAM domain and an intrinsically disordered region enables full SAMD1 function on chromatin.

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第一作者： Merle,Geller

第一单位： Institute of Molecular Biology and Tumor Research (IMT), Philipps University of Marburg, Marburg 35043, Germany.

作者： Merle,Geller ^[1] ; Yinghua,Cao ^[2] ; Clara,Simon ^[1] ; Bastian,Stielow ^[1] ; Jingfei,Xu ^[2] ; Pengshuai,Wei ^[2] ; Andrea,Nist ^[3] ; Iris,Rohner ^[1] ; Lea Marie,Jeude ^[1] ; Theresa,Huber ^[1] ; Thorsten,Stiewe ^[3] ; Zhanxin,Wang ^[2] ; Robert,Liefke ^[4]

作者单位： Institute of Molecular Biology and Tumor Research (IMT), Philipps University of Marburg, Marburg 35043, Germany. ^[1] Key Laboratory of Cell Proliferation and Regulation Biology of Ministry of Education, College of Life Sciences, Beijing Normal University, Beijing 100875, China. ^[2] Genomics Core Facility, Institute of Molecular Oncology, Member of the German Center for Lung Research (DZL), Philipps University of Marburg, Marburg 35043, Germany.;Institute for Lung Health (ILH), Justus Liebig University, Giessen 35392, Germany. ^[3] Institute of Molecular Biology and Tumor Research (IMT), Philipps University of Marburg, Marburg 35043, Germany.;Department of Hematology, Oncology, and Immunology, University Hospital Giessen and Marburg, Marburg 35043, Germany. ^[4]

主题词染色质(Chromatin);人类(Humans);蛋白质多聚化(Protein Multimerization);蛋白质结合(Protein Binding);模型, 分子(Models, Molecular)

DOI 10.1093/nar/gkaf259

PMID 40183636

发布时间 2025-04-17

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