A circRNA-mRNA pairing mechanism regulates tumor growth and endocrine therapy resistance in ER-positive breast cancer.
第一作者:
Jia,Yi
第一单位:
State Key Laboratory of Cellular Stress Biology, School of Pharmaceutical Sciences, Xiamen University, Xiamen 361102, Fujian, China.;Fujian Provincial Key Laboratory of Innovative Drug Target Research, School of Pharmaceutical Sciences, Xiamen University, Xiamen 361102, Fujian, China.;Xiang An Biomedicine Laboratory, School of Pharmaceutical Sciences, Xiamen University, Xiamen 361102, Fujian, China.;Yu-Yue Pathology Scientific Research Center, Chongqing 400039, China.;Jinfeng Laboratory, Chongqing 401329, China.
作者:
主题词
人类(Humans);乳腺肿瘤(Breast Neoplasms);女(雌)性(Female);抗药性, 肿瘤(Drug Resistance, Neoplasm);细胞周期蛋白D1(Cyclin D1);动物(Animals);小鼠(Mice);细胞系, 肿瘤(Cell Line, Tumor);细胞增殖(Cell Proliferation);RNA, 信使(RNA, Messenger);受体, 雌激素(Receptors, Estrogen);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);抗肿瘤药, 激素(Antineoplastic Agents, Hormonal);他莫昔芬(Tamoxifen);小鼠, 裸(Mice, Nude)
DOI
10.1073/pnas.2420383122
PMID
40233410
发布时间
2025-04-19
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