换一批CD163<sup>+</sup> macrophages attenuate pressure overload-induced left ventricular systolic dysfunction and cardiac mitochondrial dysfunction via interleukin-10.
第一作者：
Wei,Ni
第一单位：
School of Medicine, Xiamen Cardiovascular Hospital, Xiamen University, Jinshan Road 2999, Xiamen, 361015, China.;Sir Run Run Shaw Hospital, School of Medicine, Zhejiang University, Qingchun East Road 3, Hangzhou, 310016, China.
作者：
医学主题词
动物(Animals);心室功能障碍, 左(Ventricular Dysfunction, Left);白细胞介素10(Interleukin-10);巨噬细胞(Macrophages);抗原, 分化, 髓单核细胞(Antigens, Differentiation, Myelomonocytic);小鼠, 基因敲除(Mice, Knockout);受体, 细胞表面(Receptors, Cell Surface);小鼠, 近交C57BL(Mice, Inbred C57BL);抗原, CD(Antigens, CD);线粒体, 心脏(Mitochondria, Heart);疾病模型, 动物(Disease Models, Animal);男(雄)性(Male);小鼠(Mice);心室功能, 左(Ventricular Function, Left);心力衰竭(Heart Failure);肌细胞, 心脏(Myocytes, Cardiac)
DOI
10.1007/s00395-025-01114-z
PMID
40343453
发布时间
2026-03-16
基金项目
2019YFE0113900/National Key Research and Development Program of China
3502Z20204005/Xiamen Medical and Health Key Project
2021QNB021/Fujian Provincial Health Technology Project
2023/Scientific Research Foundation of the Xiamen Human Resource Ministry for Returned Chinese Scholars
2024J011424/Natural Science Foundation of Fujian Province
2024J011428/Natural Science Foundation of Fujian Province
3502Z20224ZD1182/Xiamen Medical and Health Project
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Basic research in cardiology
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