Recharacterization of the Tumor Suppressive Mechanism of RSL3 Identifies the Selenoproteome as a Druggable Pathway in Colorectal Cancer.
第一作者:
Stephen L,DeAngelo
作者:
医学主题词
人类(Humans);结直肠肿瘤(Colorectal Neoplasms);硒蛋白质类(Selenoproteins);小鼠(Mice);动物(Animals);细胞系, 肿瘤(Cell Line, Tumor);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays);蛋白质组(Proteome);咔啉类(Carbolines)
DOI
10.1158/0008-5472.CAN-24-3478
PMID
40392234
发布时间
2026-07-22
基金项目
R01CA148828/Center for Cancer Research (CCR)
R01 CA148828/CA/NCI NIH HHS/United States
P30CA046592/Comprehensive Cancer Center, University of Michigan (U-M Comprehensive Cancer Center)
R01 CA245546/CA/NCI NIH HHS/United States
R01 CA262439/CA/NCI NIH HHS/United States
R01DK095201/National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
623914/Crohn's and Colitis Foundation (CCF)
R01CA262439/National Cancer Institute (NCI)
R01CA245546/National Cancer Institute (NCI)
R01CA266513/National Cancer Institute (NCI)
R01GM132129/National Institute of General Medical Sciences (NIGMS)
R01DK124384/National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
19POST34380588/American Heart Association (AHA)
CA286898/National Cancer Institute (NCI)
GM117141/National Institute of General Medical Sciences (NIGMS)
GM150581/National Institute of General Medical Sciences (NIGMS)
- 浏览1
Cancer research
2788-2804页
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