换一批Antagonizing CCR2 With Propagermanium Leads to Altered Distribution of Macrophage Subsets and Favorable Tissue Remodeling After Myocardial Infarction in Mice.
第一作者：
Kay,Weipert
第一单位：
Department of Internal Medicine I, Division of Cardiology, University Clinic Giessen, Giessen, Germany.;Department of Experimental Cardiology, Kerckhoff Heart and Thorax Center, Bad Nauheim, Germany.
作者：
医学主题词
动物(Animals);心肌梗死(Myocardial Infarction);受体, CCR2(Receptors, CCR2);小鼠, 近交C57BL(Mice, Inbred C57BL);巨噬细胞(Macrophages);疾病模型, 动物(Disease Models, Animal);心室重构(Ventricular Remodeling);男(雄)性(Male);心肌(Myocardium);锗(Germanium);时间因素(Time Factors);心室功能, 左(Ventricular Function, Left);纤维化(Fibrosis);小鼠(Mice);有机金属化合物(Organometallic Compounds);丙酸盐类(Propionates)
DOI
10.1155/cdr/8856808
PMID
40606572
发布时间
2025-07-04
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Cardiovascular therapeutics
8856808页
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