Elevated Acetylation of MFN2 is Accompanied by the Disruption of Mitochondrial Energy Metabolism and Inflammation in a Mouse Model of Depression.
第一作者:
Xiaoxian,Xie
第一单位:
Shanghai Mental Health Center, Shanghai Jiao Tong University, School of Medicine, Shanghai, 201109, China.;Department of Pharmacology, University of Oxford, Mansfield Road, OX1 3QT, Oxford, UK.
作者:
医学主题词
动物(Animals);乙酰化作用(Acetylation);线粒体(Mitochondria);炎症(Inflammation);能量代谢(Energy Metabolism);GTP磷酸水解酶类(GTP Phosphohydrolases);疾病模型, 动物(Disease Models, Animal);男(雄)性(Male);抑郁(Depression);抗衰老酶1(Sirtuin 1);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠(Mice);氧化性应激(Oxidative Stress);应激, 心理学(Stress, Psychological);线粒体蛋白质类(Mitochondrial Proteins)
DOI
10.1007/s12035-025-05222-8
PMID
40668505
发布时间
2025-10-09
- 浏览0
Molecular neurobiology
14167-14175页
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