USP47 Regulates Excitatory Synaptic Plasticity and Modulates Seizures in Murine Models by Blocking Ubiquitinated AMPAR Degradation.
第一作者:
Juan,Yang
第一单位:
Department of Neurology, the Second Affiliated Hospital of Chongqing Medical University, Chongqing, 400010, China.;Department of Neurology, Affiliated Hospital of Zunyi Medical University, Zunyi, 563000, China.
作者:
医学主题词
动物(Animals);受体, AMPA(Receptors, AMPA);神经元可塑性(Neuronal Plasticity);发作(Seizures);疾病模型, 动物(Disease Models, Animal);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠(Mice);泛素硫酯酶(Ubiquitin Thiolesterase);男(雄)性(Male);兴奋性突触后电位(Excitatory Postsynaptic Potentials);泛素化(Ubiquitination);树突棘(Dendritic Spines);海马(Hippocampus)
DOI
10.1007/s12264-025-01441-0
PMID
40716012
发布时间
2025-10-07
- 浏览0
Neuroscience bulletin
2025年41卷10期
1805-1823页
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