换一批Ceftriaxone ameliorates tau phosphorylation and mislocalization in APP/PS1 AD mice by inhibiting endoplasmic reticulum stress.
第一作者：
Niu-Niu,Feng
第一单位：
Hebei Key Laboratory of Critical Disease Mechanism and Intervention, Department of Pathophysiology, Neuroscience Research Center, Hebei Medical University, 361 Zhongshan East Road, Shijiazhuang, 050017, PR China; Department of Basic Medicine, Hebei North University, 11 Zuanshi South Road, Zhangjiakou, 075000, PR China.
作者：
医学主题词
动物(Animals);tau蛋白质类(tau Proteins);磷酰化(Phosphorylation);小鼠(Mice);海马(Hippocampus);小鼠, 转基因(Mice, Transgenic);淀粉样β蛋白前体(Amyloid beta-Protein Precursor);阿尔茨海默病(Alzheimer Disease);头孢曲松(Ceftriaxone);小鼠, 近交C57BL(Mice, Inbred C57BL);早老素1(Presenilin-1);男(雄)性(Male);疾病模型, 动物(Disease Models, Animal);神经保护药(Neuroprotective Agents);突触体(Synaptosomes)
DOI
10.1016/j.neuropharm.2025.110600
PMID
40716511
发布时间
2025-09-05
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Neuropharmacology
110600页
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