Innate immune mechanisms hijacked by leukemia-initiating stem cells for selective advantage and immune evasion in Ptpn11-associated JMML.
第一作者:
Hong,Zheng
第一单位:
Department of Pediatrics, Aflac Cancer & Blood Disorders Center, Winship Cancer Institute, Children's Healthcare of Atlanta, Emory University School of Medicine, Atlanta, GA, USA; Department of Pathophysiology, Anhui Medical University, Hefei, China. Electronic address: zhenghong@ahmu.edu.cn.
作者:
医学主题词
动物(Animals);免疫, 先天(Immunity, Innate);蛋白质酪氨酸磷酸酶, 非受体11型(Protein Tyrosine Phosphatase, Non-Receptor Type 11);小鼠(Mice);白血病, 粒-单核细胞, 幼年型(Leukemia, Myelomonocytic, Juvenile);肿瘤干细胞(Neoplastic Stem Cells);免疫逃避(Immune Evasion);钙粒蛋白A(Calgranulin A);钙粒蛋白B(Calgranulin B);突变(Mutation);信号传导(Signal Transduction);小鼠, 近交C57BL(Mice, Inbred C57BL);人类(Humans)
DOI
10.1016/j.celrep.2025.116087
PMID
40748755
发布时间
2025-09-16
- 浏览0
Cell reports
116087页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文


换一批



