Crosstalk between macrophages and fibroblasts contributes to inflammation and damage in giant cell arteritis.
第一作者:
Shuang,Xu
第一单位:
Department of Rheumatology and Clinical Immunology, University Medical Center Groningen, University of Groningen, Groningen, the Netherlands.
作者:
医学主题词
人类(Humans);巨细胞动脉炎(Giant Cell Arteritis);成纤维细胞(Fibroblasts);巨噬细胞(Macrophages);男(雄)性(Male);女(雌)性(Female);老年人(Aged);腱糖蛋白(Tenascin);粒细胞巨噬细胞集落刺激因子(Granulocyte-Macrophage Colony-Stimulating Factor);颞动脉(Temporal Arteries);中年人(Middle Aged);细胞, 培养的(Cells, Cultured);炎症(Inflammation);巨噬细胞集落刺激因子(Macrophage Colony-Stimulating Factor);主动脉(Aorta);老年人, 80以上(Aged, 80 and over);细胞间通讯(Cell Communication);细胞因子类(Cytokines);病例对照研究(Case-Control Studies);基质金属蛋白酶3(Matrix Metalloproteinase 3)
DOI
10.1093/rheumatology/keaf408
PMID
40795165
发布时间
2026-03-28
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