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Genome-wide association study of neuropathic pain phenotypes implicates loci involved in neural cell adhesion, channels, collagen matrix formation, and immune regulation.

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第一作者： Richard,Packer

第一单位： Department of Population Health Sciences, University of Leicester, Leicester, United Kingdom.;Leicester NIHR Biomedical Research Centre, Glenfield Hospital, Leicester, United Kingdom.;Leicester British Heart Foundation Centre of Research Excellence, Leicester, United Kingdom.

作者： Richard,Packer ^[1] ; Kayesha,Coley ^[2] ; Alexander T,Williams ^[2] ; Nick,Shrine ^[2] ; Abril G,Izquierdo ^[2] ; Jing,Chen ^[2] ; Chiara,Batini ^[2] ; Mikko,Marttila ^[3] ; Balasubramanya S,Rao ^[4] ; Raymond,Bratty ^[4] ; Frank,Dudbridge ^[5] ; William,Hennah ^[6] ; Martin D,Tobin ^[1]

作者单位： Department of Population Health Sciences, University of Leicester, Leicester, United Kingdom.;Leicester NIHR Biomedical Research Centre, Glenfield Hospital, Leicester, United Kingdom.;Leicester British Heart Foundation Centre of Research Excellence, Leicester, United Kingdom. ^[1] Department of Population Health Sciences, University of Leicester, Leicester, United Kingdom. ^[2] Orion Pharma, Espoo, Finland. ^[3] Orion Pharma, Nottingham, United Kingdom. ^[4] Department of Population Health Sciences, University of Leicester, Leicester, United Kingdom.;Leicester NIHR Biomedical Research Centre, Glenfield Hospital, Leicester, United Kingdom. ^[5] Orion Pharma, Espoo, Finland.;Neuroscience Center, HiLIFE, University of Helsinki, Helsinki, Finland. ^[6]