Molecular hydrogen mitigates acetaminophen-induced liver injury and enhances the effects of N-acetylcysteine in diabetic mice.
第一作者:
Naomi,Kamimura
第一单位:
Laboratory for Clinical Research, Collaborative Research Center, Nippon Medical School, Tokyo, Japan; Department of Biochemistry and Cell Biology, Graduate School of Medicine, Nippon Medical School, Tokyo, Japan. Electronic address: nanakash@nms.ac.jp.
作者:
医学主题词
动物(Animals);醋氨酚(Acetaminophen);乙酰半胱氨酸(Acetylcysteine);氢(Hydrogen);小鼠(Mice);氧化性应激(Oxidative Stress);成纤维细胞生长因子(Fibroblast Growth Factors);男(雄)性(Male);糖尿病, 实验性(Diabetes Mellitus, Experimental);肝(Liver);小鼠, 转基因(Mice, Transgenic);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);bcl-2相关X蛋白质(bcl-2-Associated X Protein);丙氨酸转氨酶(Alanine Transaminase);天冬氨酸氨基转移酶类(Aspartate Aminotransferases);小鼠, 近交C57BL(Mice, Inbred C57BL)
DOI
10.1016/j.jnutbio.2025.110176
PMID
41207540
发布时间
2026-01-04
- 浏览1
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文


换一批



