首页> Experimental eye research> Progressive rod bipolar cell loss, ubiquitin accumulation, and subretinal drusenoid deposits in Cln3Δex7/8 mice: New insights into juvenile neuronal ceroid lipofuscinosis pathogenesis.

Progressive rod bipolar cell loss, ubiquitin accumulation, and subretinal drusenoid deposits in Cln3Δex7/8 mice: New insights into juvenile neuronal ceroid lipofuscinosis pathogenesis.

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第一作者： Tommi,Torsti

第一单位： School of Pharmacy, University of Eastern Finland, Kuopio, Finland. Electronic address: tommi.torsti@uef.fi.

作者： Tommi,Torsti ^[1] ; Toni,Tamminen ^[2] ; Elisa,Toropainen ^[3] ; Aaro,Jalkanen ^[4] ; Anu,Kauppinen ^[4] ; Kai,Kaarniranta ^[5] ; Markus M,Forsberg ^[4]

作者单位： School of Pharmacy, University of Eastern Finland, Kuopio, Finland. Electronic address: tommi.torsti@uef.fi. ^[1] Department of Ophthalmology, Institute of Clinical Medicine, University of Eastern Finland, Finland. ^[2] School of Pharmacy, University of Eastern Finland, Kuopio, Finland; Department of Ophthalmology, Institute of Clinical Medicine, University of Eastern Finland, Finland. ^[3] School of Pharmacy, University of Eastern Finland, Kuopio, Finland. ^[4] Department of Ophthalmology, Kuopio University Hospital, Finland; Department of Ophthalmology, Institute of Clinical Medicine, University of Eastern Finland, Finland. ^[5]