USP9X suppression attenuates NLRP3 inflammasome activation and ameliorates neuroinflammatory phenotypes with motor function recovery in murine models.
第一作者:
Yu,Guo
第一单位:
Anhui Engineering Research Center for Neural Regeneration Technology and Medical New Materials, Bengbu Medical University, Bengbu 233000, China; School of Laboratory Medicine, Bengbu Medical University, Bengbu 233000 Anhui, China. Electronic address: guoyu@bbmu.edu.cn.
作者:
医学主题词
动物(Animals);泛素硫酯酶(Ubiquitin Thiolesterase);炎性体(Inflammasomes);小鼠(Mice);疾病模型, 动物(Disease Models, Animal);男(雄)性(Male);小鼠, 近交C57BL(Mice, Inbred C57BL);小神经胶质细胞(Microglia);帕金森病(Parkinson Disease);泛素蛋白连接酶类(Ubiquitin-Protein Ligases);脂多糖类(Lipopolysaccharides);功能恢复(Recovery of Function);炎症(Inflammation);运动活动(Motor Activity);表型(Phenotype);脑(Brain)
DOI
10.1016/j.bbi.2025.106215
PMID
41381011
发布时间
2026-01-22
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