首页> FASEB journal : official publication of the Federation of American Societies for Experimental Biology> GLCCI1-Deficiency Facilitates Persistent Eosinophilic Lung Inflammation in Asthma via Upregulation of CCL5.

GLCCI1-Deficiency Facilitates Persistent Eosinophilic Lung Inflammation in Asthma via Upregulation of CCL5.

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第一作者： Huan,Ge

第一单位： Department of Respiratory Medicine, National Key Clinical Specialty, Branch of National Clinical Research Center for Respiratory Disease, Xiangya Hospital, Central South University, Changsha, China.;National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan, China.

作者： Huan,Ge ^[1] ; Yuanyuan,Jiang ^[1] ; Xinyue,Hu ^[1] ; Lisha,Luo ^[1] ; Chendong,Wu ^[1] ; Shuanglinzi,Deng ^[1] ; Jiale,Tang ^[1] ; Runjin,Cai ^[1] ; Xiaoxiao,Gong ^[1] ; Guo,Wang ^[1] ; Xuemei,Chen ^[1] ; Yunbing,Jia ^[1] ; Xiaozhao,Li ^[2] ; Juntao,Feng ^[1]

作者单位： Department of Respiratory Medicine, National Key Clinical Specialty, Branch of National Clinical Research Center for Respiratory Disease, Xiangya Hospital, Central South University, Changsha, China.;National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, Hunan, China. ^[1] Department of Nephrology, Xiangya Hospital, Central South University, Changsha, Hunan, China. ^[2]

关键词 AKT pathway CCL5 GLCCI1 asthma eosinophils glucocorticoids