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Inflammation-mediated function enhancement of K<sub>Na</sub> channels promotes network hyperactivity in the peripheral cortex following TBI.

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第一作者： Lei,Sun

第一单位： Department of Neurology, Zhengzhou University People's Hospital, Zhengzhou, China; Department of Neurology, Henan Provincial People's Hospital, Zhengzhou, China. Electronic address: sunlei020721@163.com.

作者： Lei,Sun ^[1] ; Ru,Liu ^[2] ; Xiong,Han ^[3] ; Na,Wang ^[3] ; Ting,Zhao ^[3] ; Yanan,Chen ^[3] ; Jianping,Wu ^[4] ; Junfan,Chen ^[5] ; Qun,Wang ^[6]

作者单位： Department of Neurology, Zhengzhou University People's Hospital, Zhengzhou, China; Department of Neurology, Henan Provincial People's Hospital, Zhengzhou, China. Electronic address: sunlei020721@163.com. ^[1] Department of Neurology, The First Affiliated Hospital of Shandong First Medical University, Ji'nan, China. ^[2] Department of Neurology, Zhengzhou University People's Hospital, Zhengzhou, China; Department of Neurology, Henan Provincial People's Hospital, Zhengzhou, China. ^[3] Beijing Tiantan Hospital, Capital Medical University, Beijing, China; Advanced Innovation Center for Human Brain Protection, Capital Medical University, Beijing, China. ^[4] The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China. Electronic address: evanchen@surgery.cuhk.edu.hk. ^[5] Beijing Tiantan Hospital, Capital Medical University, Beijing, China; The First Affiliated Hospital of Zhengzhou University, Zhengzhou, China; China National Clinical Research Center for Neurological Diseases, Beijing, China. Electronic address: wangq@ccmu.edu.cn. ^[6]

关键词 Epileptogenesis Hyperactive network K(Na) channels NF-κB Post-traumatic epilepsy Traumatic brain injury

DOI 10.1016/j.eplepsyres.2026.107751

PMID 41653872

发布时间 2026-02-07

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