A gene therapy strategy using a transcription factor decoy of the E2F binding site inhibits smooth muscle proliferation in vivo.
第一作者:
R,Morishita
第一单位:
Division of Cardiovascular Medicine, Falk Cardiovascular Research Center, Stanford University School of Medicine, CA 94305-5246, USA.
作者:
主题词
动物(Animals);主动脉(Aorta);碱基序列(Base Sequence);结合部位(Binding Sites);CDC2蛋白激酶(CDC2 Protein Kinase);载体蛋白质类(Carrier Proteins);细胞周期蛋白质类(Cell Cycle Proteins);细胞分裂(Cell Division);细胞核(Cell Nucleus);细胞, 培养的(Cells, Cultured);DNA引物(DNA Primers);DNA结合蛋白质类(DNA-Binding Proteins);药物载体(Drug Carriers);E2F转录因子类(E2F Transcription Factors);基因, myc(Genes, myc);脂质体(Liposomes);分子序列数据(Molecular Sequence Data);肌, 平滑, 血管(Muscle, Smooth, Vascular);寡脱氧核糖核苷酸类(Oligodeoxyribonucleotides);聚合酶链反应(Polymerase Chain Reaction);增殖细胞核抗原(Proliferating Cell Nuclear Antigen);启动区, 遗传(Promoter Regions, Genetic);大鼠(Rats);视网膜母细胞瘤结合蛋白1(Retinoblastoma-Binding Protein 1);转录因子DP1(Transcription Factor DP1);转录因子(Transcription Factors);转录激活(Transcriptional Activation);转染(Transfection)
DOI
10.1073/pnas.92.13.5855
PMID
7597041
发布时间
2019-05-01
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