Eosinophil cationic granule proteins impair thrombomodulin function. A potential mechanism for thromboembolism in hypereosinophilic heart disease.
第一作者:
A,Slungaard
第一单位:
Department of Medicine, University of Minnesota Medical School Minneapolis 55455.
作者:
医学主题词
老年人(Aged);血蛋白质类(Blood Proteins);心内膜炎(Endocarditis);内皮(Endothelium);嗜酸细胞颗粒蛋白质类(Eosinophil Granule Proteins);嗜酸粒细胞增多(Eosinophilia);心脏病(Heart Diseases);人类(Humans);男(雄)性(Male);膜糖蛋白类(Membrane Glycoproteins);钾(Potassium);蛋白质C(Protein C);受体, 细胞表面(Receptors, Cell Surface);受体, 凝血酶(Receptors, Thrombin);核糖核酸酶类(Ribonucleases);凝血酶(Thrombin);血栓栓塞(Thromboembolism)
DOI
10.1172/JCI116382
PMID
8386194
发布时间
2022-03-31
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