Cholecystokinin stimulates formation of shc-grb2 complex in rat pancreatic acinar cells through a protein kinase C-dependent mechanism.
第一作者:
A,Dabrowski
第一单位:
Department of Physiology, University of Michigan, Ann Arbor, Michigan 48109-0622, USA.
作者:
医学主题词
3T3细胞(3T3 Cells);衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);胆囊收缩素(Cholecystokinin);表皮生长因子(Epidermal Growth Factor);GRB2衔接蛋白质(GRB2 Adaptor Protein);GTP结合蛋白质类(GTP-Binding Proteins);鸟嘌呤核苷酸交换因子类(Guanine Nucleotide Exchange Factors);小鼠(Mice);胰腺(Pancreas);磷酰化(Phosphorylation);蛋白激酶C(Protein Kinase C);蛋白质类(Proteins);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 胆囊收缩素(Receptors, Cholecystokinin);酪氨酸(Tyrosine);ras鸟嘌呤核苷酸交换因子类(ras Guanine Nucleotide Exchange Factors)
DOI
10.1074/jbc.271.43.27125
PMID
8900204
发布时间
2021-02-10
基金项目
DK34171/DK/NIDDK NIH HHS/United States
R01 DK041122/DK/NIDDK NIH HHS/United States
DK41122/DK/NIDDK NIH HHS/United States
P30 DK034933/DK/NIDDK NIH HHS/United States
DK41225/DK/NIDDK NIH HHS/United States
R01 DK034171/DK/NIDDK NIH HHS/United States
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