Signaling through the ARK tyrosine kinase receptor protects from apoptosis in the absence of growth stimulation.
第一作者:
P,Bellosta
第一单位:
Department of Microbiology and the Kaplan Cancer Center, New York University School of Medicine, New York 10016, USA.
作者:
医学主题词
3T3细胞(3T3 Cells);动物(Animals);细胞凋亡(Apoptosis);CHO细胞(CHO Cells);细胞分裂(Cell Division);仓鼠亚科(Cricetinae);培养基, 无血清(Culture Media, Serum-Free);DNA复制(DNA Replication);生长物质(Growth Substances);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);癌基因蛋白质类(Oncogene Proteins);磷酰化(Phosphorylation);蛋白质加工, 转译后(Protein Processing, Post-Translational);蛋白质类(Proteins);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-myc(Proto-Oncogene Proteins c-myc);受体蛋白质酪氨酸激酶类(Receptor Protein-Tyrosine Kinases);重组融合蛋白质类(Recombinant Fusion Proteins);信号传导(Signal Transduction);星状孢子素(Staurosporine);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);紫外线(Ultraviolet Rays)
DOI
10.1038/sj.onc.1201419
PMID
9395235
发布时间
2022-12-07
- 浏览18
Oncogene
2387-97页
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