Activation of a metabotropic glutamate receptor and protein kinase C reduce the extent of inactivation of the K+ channel Kv1.1/Kvbeta1.1 via dephosphorylation of Kv1.1.
第一作者:
M,Levy
第一单位:
Department of Physiology and Pharmacology, Sackler School of Medicine, Tel-Aviv University, 69978 Ramat Aviv, Israel.
作者:
主题词
氨基酸序列(Amino Acid Sequence);动物(Animals);细胞膜(Cell Membrane);导电性(Electric Conductivity);电生理学(Electrophysiology);离子通道闸门(Ion Channel Gating);Kv1.1钾通道(Kv1.1 Potassium Channel);分子序列数据(Molecular Sequence Data);卵母细胞(Oocytes);膜片钳术(Patch-Clamp Techniques);磷酰化(Phosphorylation);钾通道(Potassium Channels);钾通道, 电压门控(Potassium Channels, Voltage-Gated);蛋白质结合(Protein Binding);蛋白激酶C(Protein Kinase C);受体, 亲代谢性谷氨酸盐(Receptors, Metabotropic Glutamate);信号传导(Signal Transduction);十四酰佛波乙酯(Tetradecanoylphorbol Acetate);光滑爪蟾(Xenopus laevis)
DOI
10.1074/jbc.273.11.6495
PMID
9497384
发布时间
2021-02-09
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