Burn wound infection-induced myeloid suppression: the role of prostaglandin E2, elevated adenylate cyclase, and cyclic adenosine monophosphate.
第一作者:
R L,Gamelli
第一单位:
Loyola University Medical Center, Shock Trauma Institute and Department of Surgery, Maywood, Illinois 60153, USA.
作者:
主题词
动物(Animals);骨髓(Bone Marrow);烧伤(Burns);环AMP(Cyclic AMP);地诺前列酮(Dinoprostone);疾病模型, 动物(Disease Models, Animal);大肠杆菌感染(Escherichia coli Infections);粒细胞(Granulocytes);造血干细胞(Hematopoietic Stem Cells);免疫耐受(Immune Tolerance);巨噬细胞, 腹膜(Macrophages, Peritoneal);男(雄)性(Male);小鼠(Mice);小鼠, 近交系(Mice, Inbred Strains);伤口感染(Wound Infection)
DOI
10.1097/00005373-199803000-00008
PMID
9529173
发布时间
2019-07-11
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The Journal of trauma
469-74页
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