The ability of positive transcription elongation factor B to transactivate human immunodeficiency virus transcription depends on a functional kinase domain, cyclin T1, and Tat.
第一作者:
K,Fujinaga
第一单位:
Departments of Medicine, Microbiology, and Immunology, Howard Hughes Medical Institute, University of California, San Francisco, San Francisco, California 94143-0703, USA.
作者:
主题词
细胞系, 转化(Cell Line, Transformed);细胞周期蛋白T(Cyclin T);细胞周期蛋白质依赖激酶类(Cyclin-Dependent Kinases);细胞周期蛋白类(Cyclins);基因产物, rev(Gene Products, rev);基因产物, tat(Gene Products, tat);HIV长末端重复序列(HIV Long Terminal Repeat);HIV-1(HIV-1);人类(Humans);肽延伸因子(Peptide Elongation Factors);阳性转录延伸因子B(Positive Transcriptional Elongation Factor B);转录, 遗传(Transcription, Genetic);转录激活(Transcriptional Activation);rev基因产物, 人免疫缺陷病毒(rev Gene Products, Human Immunodeficiency Virus);tat基因产物, 人免疫缺陷病毒(tat Gene Products, Human Immunodeficiency Virus)
DOI
10.1128/JVI.72.9.7154-7159.1998
PMID
9696809
发布时间
2021-12-03
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Journal of virology
7154-9页
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