Bax-mediated mitochondrial outer membrane permeabilization (MOMP), distinct from the mitochondrial permeability transition, is a key mechanism in diclofenac-induced hepatocyte injury: Multiple protective roles of cyclosporin A.

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作者： Siu WP ^[1] ; Pun PB ; Latchoumycandane C ; Boelsterli UA

作者单位： Department of Pharmacology, Yong Loo Lin School of Medicine, National University of Singapore, Singapore 117 597. inhibitors/genetics/metabolism ^[1]

关键词 Anti-Inflammatory Agents Non-Steroidal Apoptosis BH3 Interacting Domain Death Agonist Protein 消炎药非甾类脱噬作用钙细胞系螯合剂环孢菌素细胞保护二氯酚酸

发布时间 2013-11-20

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Toxicology and Applied Pharmacology

2008年227卷3期

451-461页

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Bax-mediated mitochondrial outer membrane permeabilization (MOMP), distinct from the mitochondrial permeability transition, is a key mechanism in diclofenac-induced hepatocyte injury: Multiple protective roles of cyclosporin A.

Toxicology and Applied Pharmacology

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Toxicology and Applied Pharmacology

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Bax-mediated mitochondrial outer membrane permeabilization (MOMP), distinct from the mitochondrial permeability transition, is a key mechanism in diclofenac-induced hepatocyte injury: Multiple protective roles of cyclosporin A.

Toxicology and Applied Pharmacology

相似文献

Toxicology and Applied Pharmacology

相关期刊

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