PI3Kδ inhibitor, GS-1101 (CAL-101), attenuates pathway signaling, induces apoptosis, and overcomes signals from the microenvironment in cellular models of Hodgkin lymphoma

导出原文传递

翻译打印收藏纠错

作者： Meadows,S.A. ^[1] ; Vega,F. ^[2] ; Kashishian,A. ^[3] ; Johnson,D. ^[4] ; Diehl,V. ; Miller,L.L. ; Younes,A. ; Lannutti,B.J.

作者单位： Department of Hematopathology, University of Texas M. D. Anderson Cancer Center, Houston, TX ^[1] Department of Lymphoma/Myeloma, University of Texas M. D. Anderson Cancer Center, Houston, TX ^[2] Department of Internal Medicine, Hematology-Oncology, University of Cologne, Cologne, Germany ^[3] Gilead Sciences Inc., 199 E Blain St, Seattle, WA 98102, United States ^[4]

发布时间 2013-11-20

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Blood

2012年119卷8期

1897-1900页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

PI3Kδ inhibitor, GS-1101 (CAL-101), attenuates pathway signaling, induces apoptosis, and overcomes signals from the microenvironment in cellular models of Hodgkin lymphoma

Blood

相似文献

Blood

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

PI3Kδ inhibitor, GS-1101 (CAL-101), attenuates pathway signaling, induces apoptosis, and overcomes signals from the microenvironment in cellular models of Hodgkin lymphoma

Blood

相似文献

Blood

相关期刊

检索历史清除