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Oral treatment with CuII(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis

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第一作者: Roberts,B.R.
作者单位: Centre for Cellular and Molecular Biology, School of Life and Environmental Sciences, Deakin [1] Elemental Bio-imaging Facility, University of Technology Sydney, 2007 Broadway, NSW, Australia [2] Department of Pathology, The University of Melbourne, Parkville, 3010, VIC, Australia, AI Virtanen [3] Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Parkville, 3010 [4] School of Chemistry, The University of Melbourne, Parkville, 3010, VIC, Australia, Bio21 Institute [5] Department of Pathology, The University of Melbourne, Parkville, 3010, VIC, Australia [6] Department of Biochemistry and Biophysics, Oregon State University, Corvallis, OR 97331, United [7] ANU Medical School, ANU College of Medicine, Biology and the Environment, The Australian National [8] Department of Pathology, The University of Melbourne, Parkville, 3010, VIC, Australia, Department [9]
发布时间 2014-12-17
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The Journal of Neuroscience: The Official Journal of the Society for Neuroscience

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