Ubiquinol-10 supplementation activates mitochondria functions to decelerate senescence in senescence-accelerated mice

导出原文传递

翻译打印收藏纠错

作者： Tian,G. ^[1] ; Sawashita,J. ^[2] ; Kubo,H. ^[3] ; Nishio,S.-Y. ^[4] ; Hashimoto,S. ^[5] ; Suzuki,N. ; Yoshimura,H. ; Tsuruoka,M. ; Wang,Y. ; Liu,Y. ; Luo,H. ; Xu,Z. ; Mori,M. ; Kitano,M. ; Hosoe,K. ; Takeda,T. ; Usami,S.-I. ; Higuchi,K.

作者单位： Department of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University ^[1] QOL Division, Kaneka Corporation, Osaka, Japan ^[2] Frontier Biochemical and Medical Research Laboratories, Kaneka Corporation, Hyogo, Japan ^[3] Department of Otorhinolaryngology, Shinshu University School of Medicine, Matsumoto, Japan ^[4] Council for SAM Research, Kyoto, Japan ^[5]

发布时间 2014-08-30

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Antioxidants and redox signalling

2014年20卷16期

2606-2620页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Ubiquinol-10 supplementation activates mitochondria functions to decelerate senescence in senescence-accelerated mice

Antioxidants and redox signalling

相似文献

Antioxidants and redox signalling

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Ubiquinol-10 supplementation activates mitochondria functions to decelerate senescence in senescence-accelerated mice

Antioxidants and redox signalling

相似文献

Antioxidants and redox signalling

相关期刊

检索历史清除